So, guys, Derek, moreplacemoredates.com.

Today we're going to be talking about Jay Cutler revealing his past and current cycles as well as, more notably, his stance on TRT and cycling and common practices in the realm of anabolic exposure.

So Greg Doucett released this clip from a podcast they just did recently where he basically

gets pretty fucking transparent. Now again, how truthful he is on like the offseason dosages

and whatnot, you know, it kind of remains to be seen. Like he mentions at one point how, you know,

his peak off season would typically be like, you know, 500 milligrams testosterone, 600 milligrams ecupoise or boldenone undescelainate, as is the chemical name.

Some of you guys might know it as.

But if I was to get ready for like a little, I'll be honest, off season, I didn't take as much.

I didn't do GH.

I would take always like 500 migs a test and I would use like 600

migs of Echo Poys. Echo Poys was my favorite off season. He also talks about his growth

hormonius and how he would start at. He's gone up to like, I believe it was 12 units he mentioned

in the video. But the truth is, is like, you know, my max, I mean, of course, the GH would be added in, you know, when I would train for a contest.

And listen, the dosage, I mean, remember the first dosage was four I use a day. I may have gone up to 9, 12 on and off.

I mean, I think 9 was probably the best sitting area for me. Like, that seemed to be just like around the testosterone.

I know this, I've done a thousand a week of test, but 500 was my was my sweet spot, if that makes sense.

And how he thinks it's overrated. And it's kind of interesting to hear him say that. That's something Greg has also said in the past and something I've also said that for a lot of guys who are not like, again, anabolic response is not

the same as your response at an IGF like growth factor perspective level, if that made any

fucking logical grammatical sense. Basically growth factor production and expression downstream

of proliferative activities of it,

causing muscle cell hyperplasia, myonuclear donation of those satellite cells that have proliferated,

equating to new cells that you can then induce hypertrophy in.

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