A new theory is emerging explaining the modern obesity epidemic.

And today's show we're going to talk about the fructose survival hypothesis and draw upon the work

from Richard Johnson over at University of Colorado.

I think this phenomenon is incredibly interesting. short and we'll talk about the details

the fructose survival hypothesis highlights an evolutionarily adaptive mechanism that

gets exploited in modern society where we have ample access to hyper-palatable

ultra-process junk food.

Now, this ancient survival switch,

known as the Fruto's hypothesis,

proposes that obesity and metabolic disorders may have developed

from an overstimulation of this evolutionarily based biologic response that aims to protect animals

in advance of crisis such as a food shortage.

Now it helps when there's no water, no food for this survival switch that we're going to

talk about to kick in to help animals make water from fat and utilize these different mechanisms but again in

this hyper-palatable ultra processed food environment this is where we start to see the

fructose survival switch convert into obesity and all the associated diseases like those high blood pressure. The process is initiated by the

ingestion of fructose or by stimulating endogenous fructose production via the

poly all pathway from sugar which we're going to talk about very very soon.

Unlike other nutrients fructose reduces the active energy,

adenosine triphosphate or ATP within your cells

and pivots your cells away from making,

aerobically making energy into favoring glycolysis.

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