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Perfect. So the chief concern is going to be increased drowsiness.

Increased drowsiness. Okay. Matia, you're about to be a neurologist in training here.

You hear increased drowsiness on the page, you think just two words, what am I supposed to do with this? But as you get your hammer and

things into your white coat pocket and go to do one of your first consults, what's running through

your mind here with this chief concern? Yeah, I think it's a fascinating and kind of vague chief concern. So, you know,

in neurology, we always think about the timing and the localization, and we kind of have neither here.

Now, I'm thinking that the timing will be everything here, but just to start, I think that in terms of localization,

we should not forget that drowsings can be caused

by pretty much anything in terms of systemic things.

You know, it can be something hematological.

It can be something like the cardiovascular system.

It can be something related to the lungs, you know, if they have

COPD, they're hypercarbic, hypoxic, and so forth. So I think it's important not to forget

those things. Now, in terms of localization, we did the Norexis. Of course, drowsiness, we know that it localizes mostly to the cerebrum,

although most, if we're thinking of drowsiness as impairment of awareness or consciousness,

then it would localize either to the whole cerebrum, to both hemispheres, or to the Talamai, or to the R cerebron to both hemispheres or to the thalamide or to the to the r a s s right the palms essentially

so that's a thought now what can impair both hemisphere either they're having a massive stroke that

affects everything a basilar infarction, something like that.

Or we could think if it's more of a sub-acute problem, we could think of something toxic metabolic.

Can we go back to our hypoxia and hypercarbia and drugs and so on and so forth.

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