All right, welcome. My name is Divine. This is episode 609 of the Divine Intervention Podcasts. In today's

podcast, we're going to be addressing a topic that is pretty high yield for the US Emily Step 2,

CK and Step 3 exams. And I'm going to call this podcast A&S Drugs for Step 2 and Step 3. A&S drugs for step 2

and step 3. The thing is, if you really want to drill crazy,

crazy deep, you know, I guess that's one thing that kind of freaks people out that, hey,

you know, drilling very deep into the A&S pharmacology is too much for me, right? Because

typically for step one, you need to know a lot of details. But if you're like,

divine, what do I need to know for step two and step three with regards to A&S drugs, right? Because again,

basic sciences have started making a bigger and bigger showing on the USMLA step two, see, and step three

exam. So, hey, what do I need to know about the ANSS drugs? What A&S drugs do I need to know, right?

So we're going to capture that here. If you know what I teach you here, then you're going to be pretty

much set from an A&S drug perspective for your exams. Now, real quick, before we jump into the A&S

drugs, I think it's just helpful to discuss the different AS receptors, right? So number one, right, if you think about it,

for the most part, we have the adrenergic receptors, right? We have the adrenergic receptors, right?

So like alpha 1, beta 1, alpha 2, beta 2, we'll talk about those, right? And then there's the

mostcarinic receptors, which we'll talk about as well. All right. So the key things to know here,

right? So let's talk about alpha one, right? So alpha one, remember, they do certain critical things,

right? Number one is that you're going to find them on your blood vessels, right? They're going to

cause, when you stimulate them, you're going to get visal constriction. So if you constrict your arteries, right, you're going to cause, when you stimulate them, you're going to get visal constriction.

So if you constrict your arteries, right, you're going to get, you know, arterial visal

constriction.

That's going to increase your afterload.

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