Welcome back to the IBCC podcast. I'm here with Adam and we're going to talk about

Torsod's a paw or toward, I don't know how to pronounce that actually.

You're closer to Montreal than I am. Come on. Your French should be better there.

Let's just stick with TDP.

Got it.

Torzot.

Torzod. I like it.

All right, guys, Torsat. So today it's going to be pretty straightforward. This is a little tidbit to keep you tight in that resuscitation mode. So we're

going to cover the diagnosis, your treatment options, and then some pitfalls. Josh, is it polymorphic VT?

Is it congenital? Is it acquired? These things get confusing. If I'm in there in a crashing

patient in front of me, how am I going to approach this? So the first question, if you're looking

at an EKG, I suppose, is it really polymorphic VT or is it something totally different, like

APB with WPW, really coarse ventricular fibrillation, or hypercalimemia with a sineway pattern. I think usually you can generally get a sense of it just by looking at the EKG and kind of

being aware of those different possibilities.

For example, AFID with WPW is going to be like super, super fast.

It has like a specific look to it that's not quite torsati.

And likewise, hypercalemia with a sine wave pattern, it's going to be more broad-based. But I think it's worth at least keeping in mind that simply because something looks like polymorphic vT does not mean

that it's always going to be polymorphic vT. And then once you've kind of determined that something

is polymorphic v.T, the next question is, is that truly torsad, or is it actually non-torzod polymorphic bt. And this is a little tricky, but it's an important distinction

because sometimes people kind of equate polymorphic ventricular tachycardia with Torsad like I just

did. And that's a mistake. So don't fall into the trap. So it comes out to the clincher question here,

doesn't it? Is the QTC or the QT interval normal or prolonged. Exactly.

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