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Quick, let's get on with it. First up is Swami on the management of the patient with

ACE inhibitor or bradycinine-mediated angioidema.

We're not talking anaphylaxis here.

That's a whole different kettle of fish that we covered in detail in episode 78, live from

the EMCases course.

Swami is going to give us his approach to the scary crash airway in these patients, which

luckily is a very rare occurrence and an approach to the

non-crash airway of the angioidema patient. And I'll give you a hint. We're talking Kobe,

if you remember that, which Rubin Strayer covered in previous quick hit, and awake intubation,

which George Kovac covered on his best case ever and in the burn episode.

All right, here we go.

In recent years, I found that we spend a lot of time talking about the physiologically challenging airway.

And we should spend a lot of time talking about it.

It is more common.

It's something that we need to be really facile with. But But of course, we also see anatomically challenging airways. That's what I want to get into. One of the anatomically challenging airways that isn't so uncommon, that's angioidema. Angioedema is defined as swelling of a mucus membrane, and the tongue and the lips are the most common ones that we see. Most of the time

when we think about angioidema, we jump right to ACE inhibitor induced, but there are other

causes, and there are actually two flavors of angiodema we need to think about. The first is

...