Okay, welcome back to Behind the Knife. Our Absight series continues with part two of

a patibiliary. Like I mentioned before, we had published our Absight companion.

We're looking to potentially add to our content this year.

So if anybody has any ideas for topics for the ab site, please reach out.

You can reach us at BTK podcast at gmail.com or reach us on Twitter or Facebook. Okay, take it away Jason. All right,

let's jump right back into it. So we're gonna start off with some

hepatobiliary lesions. We'll start off with cystiastic lesions first. So Wu, when I say

collodocal cyst, what comes to mind? What are those? Tell us a little bit about

them. Yeah, so collocal Sis their ideology is

unknown but it's likely secondary to an anomalous biliary pancreatic duck

junction that can generate reflux of pancreatic enzymes. And so these patients

also tend to have a long common BP duct. Most of these are identified and

treated in early childhood.

They can cause pain, bilier obstruction, and cirrhosis.

Overall, you can break them apart into the Todani classification. And we'll go through these there's one through five.

Okay so let's just go through these systematically so this is again the todani classification

for collidoccal cyst as we said typically in child classification for Koli-Dokal Sist.

As Wu said, typically in childhood,

not really sure why they happen,

but it's that if they had to put your money on something

to be that anomalous biliop pancreatic duct with a long common

duct and there's reflux up and it causes these cysts.

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