Hey all, welcome back to the Real Life Pharmacology podcast. I'm your host, pharmacist, Eric Christensen,

and I thank you so much for listening today. Go check out Real Life Pharmacology.com. Get your free 31-page

PDF on the Top 200 drugs. Great little study guide or review if you're looking to brush up as a

practicing clinician. So again, top 200 drugs,

I lay out most important clinical pearls you should know as well as things that you're going

to actually see out in clinical practice. So simple email will get you that PDF absolutely for

free. All right, let's get into the drug of the day today, and that is

a tennelal. Brand name of this drug is to Norman. It has been around the block for quite a while.

I will say its use has declined since I first started my career, falling out of favor because it doesn't have

certain indications and maybe not so great in hypertension. But anyway, it is a beta blocker.

So that's its mechanism of action as well. It selectively blocks beta 1 receptors. So we don't need to worry as much about blocking beta 2.

Remember, beta 2 receptors are on the lungs

so that can impact the breathing and things like that.

So primarily selective for beta 1.

With that said, I always say as you escalate doses, typically with any

medication selectivity is going to decline. So there certainly is potential as we get maybe more

so to higher doses that there could be impacts on those beta 2 receptors there.

So indications, what would we use atenol for?

So historically, atrophibulation, angina, hypertension, history of MI, migraine, maybe off-label.

And there's a few other obscure ones as well. In my experience, atrophibulation

and MI are probably the two most common. Hypertension used to be more common, but it is definitely

not a preferred agent in hypertension anymore. So understanding some of the uses that it's going to be used for,

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